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Article number: AP1001.1

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Laminin

Article number: AP1001.2

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Article number: AP1003.1

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Host	Rabbit
Application	Immunohistochemistry on frozen sections (IHC-Fs),ELISA,Western Blot (WB),Immunohistochemistry (IHC),Radioimmunoassay (RIA)
Reactivity	Mouse
Alternative names	Mouse Nidogen 1

Article number: AP1003.2

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Host	Rabbit
Application	Immunohistochemistry on frozen sections (IHC-Fs),ELISA,Western Blot (WB),Immunohistochemistry (IHC),Radioimmunoassay (RIA)
Reactivity	Mouse
Alternative names	Mouse Nidogen 1

Article number: AP1004.1

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Host	Rabbit
Application	Immunohistochemistry on frozen sections (IHC-Fs),Immunohistochemistry on paraffin sections (IHC-P),ELISA,Western Blot (WB),Immunohistochemistry (IHC),Radioimmunoassay (RIA)
Reactivity	Mouse
Alternative names	Mouse Nidogen 2

Article number: AP1004.2

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Host	Rabbit
Application	Immunohistochemistry on frozen sections (IHC-Fs),Immunohistochemistry on paraffin sections (IHC-P),ELISA,Western Blot (WB),Immunohistochemistry (IHC),Radioimmunoassay (RIA)
Reactivity	Mouse
Alternative names	Mouse Nidogen 2

Article number: RC0009

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Description	Mediates ATP-dependent export of organic anions and drugs from the cytoplasm. Hydrolyzes ATP with low efficiency. Human MDR3 is not capable of conferring drug resistance. Mediates the translocation of phosphatidylcholine across the canalicular membrane of the hepatocyte. Defects in ABCB4 are the cause of progressive familial intrahepatic cholestasis type 3 (PFIC3). PFIC3 is an autosomal recessive liver disorder presenting with early onset cholestasis that progresses to cirrhosis and liver failure before adulthood. It is characterized by elevated serum gamma-glutamyltransferase levels. Defects in ABCB4 are a cause of intrahepatic cholestasis of pregnancy (ICP); also known as obstetric cholestasis. ICP is a multifactorial liver disorder of pregnancy. It presents during the second or, more commonly, the third trimestre of pregnancy with intense pruritus which becomes more severe with advancing gestation and cholestasis. Cholestasis results from abnormal biliary transport from the liver in
Host	Rabbit
Application	ELISA, Immunohistochemistry (IHC), Western Blot (WB)
Reactivity	Human, Mouse

Article number: RC0009RTU7

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Description	Mediates ATP-dependent export of organic anions and drugs from the cytoplasm. Hydrolyzes ATP with low efficiency. Human MDR3 is not capable of conferring drug resistance. Mediates the translocation of phosphatidylcholine across the canalicular membrane of the hepatocyte. Defects in ABCB4 are the cause of progressive familial intrahepatic cholestasis type 3 (PFIC3). PFIC3 is an autosomal recessive liver disorder presenting with early onset cholestasis that progresses to cirrhosis and liver failure before adulthood. It is characterized by elevated serum gamma-glutamyltransferase levels. Defects in ABCB4 are a cause of intrahepatic cholestasis of pregnancy (ICP); also known as obstetric cholestasis. ICP is a multifactorial liver disorder of pregnancy. It presents during the second or, more commonly, the third trimestre of pregnancy with intense pruritus which becomes more severe with advancing gestation and cholestasis. Cholestasis results from abnormal biliary transport from the liver in
Host	Rabbit
Application	ELISA, Immunohistochemistry (IHC), Western Blot (WB)
Reactivity	Human, Mouse

Article number: RC0327

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Description	Obesity is a major risk factor for the development of insulin resistance and type 2 diabetes. Adipose tissue secretes various bioactive molecules, referred to as adipokines, whose dysregulation can mediate changes in glucose homeostasis and inflammatory responses. Adipolin or C1qdc2/CTRP12 is an insulin-sensitizing adipokine that is abundantly expressed by fat tissues and designate this adipokine as adipolin (adipose-derived insulin-sensitizing factor). Adipolin expression in adipose tissue and plasma was reduced in obesity. Systemic administration of adipolin ameliorated glucose intolerance and insulin resistance in dietinduced obese mice. Adipolin administration also reduced macrophage accumulation and proinflammatory gene expression in the adipose tissue of obesity. Studies suggest that adipolin functions as an anti-inflammatory adipokine that exerts beneficial actions on glucose metabolism. Therefore, adipolin represents a new target molecule for the treatment of insulin resistanc
Host	Rabbit
Application	ELISA, Immunohistochemistry (IHC), Western Blot (WB)
Reactivity	Human, Mouse, Rat, Bovine, Dog (Canine), Horse,Pig (Porcine), Sheep

Article number: RM0205

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Description	The annexin family of calcium-binding proteins contains several family members that are characterized by a conserved core domain which binds phospholipids in a Ca2+-dependent manner, and a unique amino-terminal region which may confer binding specificity. Annexin family members have been implicated as regulators of such diverse processes as ion flux, endocytosis, exocytosis and cellular adhesion. Annexin A10, also known as ANX14 or ANXA10, is a 324 amino acid protein that contains four Annexin domains and may be involved in the regulation of cellular growth and signal transduction pathways throughout the cell. The gene encoding Annexin A10 maps to human chromosome 4, which encodes nearly 6% of the human genome and has the largest gene deserts (regions of the genome with no protein encoding genes) of all of the human chromosomes. (Shipping Cost: €200.00)
Host	Rabbit
Application	Immunohistochemistry (IHC), Western Blot (WB)
Reactivity	Human, Mouse, Rat